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Intragroup comparisons were performed across days using repeated-measures ANOVA, and the results are summarized in Fig

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Regulates Brain Development Pathways: NAC can reduce overactivity in the Notch-1/Hes-1 pathway, which may play a role in brain development disruptions seen in ASD[3]

The TP53 mut AML clones had consistently higher cell viability in response to AraC than the TP53 WT AML clone, M14-WT1, indicating that the TP53 mut AML clones were more chemoresistant (Fig